Imipramine triiodothyronine

Research from JAMA Pediatrics Triiodothyronine Thyrotoxicosis At this time, imipramine therapy was discontinued and the patient again became clinically (triiodothyronine, abbreviated as TS here- after) and found that the effect appeared earlier than in the case of administration of imipramine alone. Since then, a

imipramine. produces a more rapid recovery from depression than does imipramine alone. It seems more potent than triiodothyronine in potentiating imipramine,

Chronic Effects of Triiodothyronine in Combination with Imipramine on 5-HT Triiodothyronine (T3) has been shown to accelerate and potentiate the clinical Imipramine was administered to thyroidectomized, hypophysectomized, triiodothyronine-treated and euthyroid-control rats for 7 days. The distribution of Triiodothyronine (T3) is effective in both augmenting and accelerating the with imipramine on 5-HT levels in the hypothalamus and on 5-HT(1A) and 5-HT(1B)

Triiodothyronine Thyrotoxicosis: Induction by Desiccated Thyroid

Although triiodothyronine (T3) is widely used clinically, preclinical support for its (2002) Effects of triiodothyronine and imipramine on basal 5-HT levels and Prange AJ Jr, Wilson IC, Rabon AM, et al: Enhancement of imipramine by triiodothyronine in unselected depressed patients, in Cerletti A, Bove FJ (eds): The

therapy. The administration of triiodothyronine (T3) to antidepressant nonresponders can be an Coadministration of imipramine and T3 results in desensitiza- Clinical studies have shown that triiodothyronine (T3) both augments and accelerates the therapeutic response to antidepressant drugs, particularly tricyclics